Dave Feldman, a software engineer, found himself at risk for a heart attack. So he took science into his own hands with an N=1 trial…on himself.

What he learned will make us question everything WE learned about cholesterol.

Watch the Facebook Live video and lend your voice in the comments. SHARE please!

And go to Dave’s website to learn more and contribute your own data.


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  • Signe Gad

    Great show! I appreciate Dave Feldmann´s work and ZdoggMD´s too for sharing controversial stuff like this. Agree, the insight is nördy, but it ought to be mainstream. For someone who doesn´t have English as first language, I recommend to read the book “Put Your Heart In Your Mouth” by Dr. Natasha Campbell-McBride before watching. I agree 100% with the critisicm of most research in nutrition. Generally, the studies are close to ridiculous, as typical results are based on the average of the participants memory, and a naive trust, that participant´s subjective reports match reality. Also lack of knowledge about the difference in fats makes a huge number of research in nutrition useless. “Rats fed with McDonalds food….blabla…. results in such and such disease…..this means that fatty food is bad.” No it doesn´t.

  • Epador

    Fun Presentation:

    I have considered fasting glucose, triglycerides and LDL as markers of lifestyle and exercise issues for those on a typical western diet. (Calling this an American diet is xenophobic). As Dave notes, those on the ketogenic diets have a different explanation for the lipid measurements. I have two concerns: once atherosclerosis, vessel narrowing and end organ damage is established, the high triglyceride levels may not be safe; if reducing inflammation is important, would higher doses of aspirin above the baby aspirin 81 mg dose to 1-2k mg a day should have benefit as well, but its not been demonstrated in studies.

  • RockyBob

    I have to admit I stopped watching at the point someone mentioned their 329 mg/dl lipid level.

    Consider my situation: I’m 71 years old, have Heterozygous Familial Hypercholesterolemia, HeFH, (first diagnosed around 1967), and have almost never had a total serum cholesterol below 300 mg/dl, even with extreme meds, and for the last eleven years my total cholesterol has been above 600, and my “bad” LDL has been above 500 mg/dl. I should have been dead al long time ago. Around 15 years ago I had my coronary arteries examined by Electron Beam Computed Tomography (EBCT) and discovered, amazingly, that I had no coronary artery calcifications. None. Score = zero. I’ve had the test done five times and the last one again was still zero. I’ve never had any symptom of any kind of heart issue. Nobody really knows why and very few are even very interested, even though I send researchers all the data. My mother had the same HeFH as does my older sister (74 years old). Neither of them ever had/has any heart issues either.

    So imagine, if you can, what it feels like to hear people talking, ad nauseam, about 30 point drops in their lipid levels, exercising this way or that, eating just the right things, avoiding this thing or that, all to get lipid levels down. My suggestion is this: find a hospital that can actually examine the state of your coronary arteries (I strongly recommend EBCT, although there are other techniques that are more invasive and/or subject you to much more radiation). Only after learning the actual state of one’s arteries can you make an informed decision about what to do. Before considering any cholesterol lowering medication please look up the website thennt.com and search for statins. The paltry benefit should discourage most anyone.

    • Colin Drew

      You have an interesting history and one that backs up my belief that monitoring insulin is more important than cholesterol.
      Good luck

  • Karie Randall

    If LDL is not the whole answer- how do we change practice? We can only use the ASCVD risk calculator until there are studies to support a change in practice.

    How do we measure oxidative stress? How do we help our patients and ourselves change diets and lifestyles to decrease oxidative stress to limit the LDL from actually depositing? What do we do for those who already have damage? Since EBCT is not a option for many without a life threatening diagnosis (or great insurance/ deep pockets) is there anything we can do to estimate or determine artery health?
    How can change the tide for young people- to reduce risk for cvd?